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reported an increase in the protein-bound form of glutathione in human blood after oral administration, while glutathione in the deproteinized fraction did not change [12]

However, this protective function prevents the majority of small molecule drugs and macromolecules (such as peptides, proteins, and gene-based drugs) entering the brain from the bloodstream, severely limiting the treatment of central nervous system (CNS) disorders [4]

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That is different from proving DSIP itself improves post-exercise repair

All reactions were run in triplicate for at least three independent experiments